The core mechanism of action of teriprexin is to mimic the function of antidiuretic hormone (vasopressin) in the body, primarily acting on V1 receptors to cause vasoconstriction in visceral blood vessels. This effect results in two key benefits:
Reducing portal venous pressure
Reducing visceral blood flow
In patients with portal hypertension, increased visceral blood flow exacerbates varicose veins, a problem effectively alleviated by teriprexin through vasoconstriction.
Furthermore, in hepatorenal syndrome, patients often experience a paradoxical state of systemic vasodilation but insufficient renal blood flow. Teriprexin improves renal function by "redistributing blood flow," increasing renal perfusion.
